![]() ![]() Acute RRD is seen as an oedematous folded retina with loss of the normal retinal transparency. Diagnosis: RRD is often, but not universally, associated with symptoms of flashes of light (retinal photopsia), visual floaters, and peripheral and usually progressive visual field loss. This review considers only acute progressive RRD. Asymptomatic and non-progressive chronic retinal detachment can also occur. Retinal detachments can also be a mixture of two or more of the above types. ![]() Rarer causes of retinal detachment include tractional retinal detachment, secondary to fibrous tissue on the surface of the retina exudative retinal detachment, as a result of choroidal tumours that produce increased fluid flow through the subretinal space and ocular inflammatory conditions. Retinal breaks can also develop as a result of trauma to and inflammation of the eye examples include retinal dialysis, which is typically secondary to blunt trauma, and tears associated with retinal necrosis, resulting from trauma or inflammation. Lattice degeneration can lead to the formation of circular retinal holes, which are typically referred to as atrophic holes. Retinal breaks that develop from a tear in the retina at the time of posterior vitreous detachment (PVD) are usually referred to as retinal tears. Retinal break refers to a full-thickness defect in the neurosensory retina. Rhegmatogenous retinal detachment (RRD) is the most common form of retinal detachment, where a retinal 'break' allows the ingress of fluid from the vitreous cavity to the subretinal space, resulting in retinal separation. ![]() Direct apposition of the retina to the RPE is essential for normal retinal function, and retinal detachment involving the foveal centre leads to profound loss of vision in the affected eye. Retinal detachment can be defined as the separation of the neurosensory retina from the underlying retinal pigment epithelium (RPE). ![]()
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